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Intracellular signal transduction from many membrane-associated receptors is redox-regulated by mechanisms involving the TrxR1 system

Oxidized arachidonic and adrenic PEs navigate cells to ferroptosis

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Long-term CFU assays showed that NAC, unlike all the cell death inhibitors, ion chelators, and cathepsin inhibitors tested herein, prevented the cytotoxic effects of DC661 in A375P, RKO, MIA PaCa-2, B16F10, and MC38 cells (Figure 5C and Supplemental Figure 7C)